SNP array distal 16p11.2 microdeletion neuropsychological phenotype social cognition. It is concluded that microdeletion syndromes are not exclusively related to intellectual disabilities and genetic testing is of putative relevance for the understanding of neuropsychiatric and neuropsychological phenomena. Willem Verbeeck America’s PlaygroundBook design and art direction for New York based photographer Willem Verbeeck's book America's Playground. Microarray analysis demonstrated a de novo 970 kb 16p11.2 BP1-BP4 microdeletion that can be regarded as explanatory for his behavioral profile.
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This more centromeric deletion is most strongly related to autism spectrum susceptibility and is functionally different from the more distal 16p12.2p11.2 region, which includes the so-called atypical 16p11.2 BP2-BP3 deletion (approximately 220 kb) presenting with developmental delay, behavioral problems and mild facial dysmorphisms. × Here is the report of the Instagram profile of willemverb ( full name: Willem Verbeeck). See the complete profile on LinkedIn and discover Willem’s connections and jobs at similar companies. Willem has 1 job listed on their profile.
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These phenotypes are often related to a proximal 16p11.2 deletion of approximately 600 kb (BP4-BP5) that includes the SH2B1 gene that is reported to be causative for morbid obesity. View Willem Verbeeck’s profile on LinkedIn, the world’s largest professional community. Early 2001 a meeting with Johnny Verbeeck was arranged, and within less than 15 minutes there was already a serious deal. The 16p11.2 microdeletion syndrome is characterized by a wide range of phenotypic expressions and is frequently associated with developmental delay, symptoms from the autism spectrum, epilepsy, congenital anomalies, and obesity. Willem Kelders father, Jan Kelders was born at Boxmeer as the second son of Wim Kelders.
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